About Us

• Our laboratory investigates the cellular and molecular mechanisms that drive inflammation and fibrosis in cardiovascular diseases, with a particular focus on how immune cells and intercellular communication shape disease progression and tissue remodeling. Using transcriptomics, advanced in vitro and in vivo disease models, as well as an abroad cellular and molecular assays, we aim to uncover how inflammatory and fibrotic networks orchestrate both injury and repair within the heart and vasculature.

• A major area of our research centers on neutrophils and NETosis, exploring how these processes influence vascular and valvular pathology, regulate fibroblast activation, and interact with other immune and vascular cell populations. We investigate the evolution of inflammatory processes and signaling during atherosclerosis, myocardial infarction, and valvular calcification, as well as the role of maladaptive communication pathways in contributing to excessive extracellular matrix deposition and tissue stiffening.

• By integrating immunology, inter-molecular and intercellular communication, and cardiovascular pathology, our goal is to identify key mechanistic drivers of inflammation and fibrosis and to identify therapeutic targets to restore balanced tissue remodeling and cardiac function.

• We welcome motivated, curious, and collaborative young scientists to join us in advancing the understanding of immune–cardiovascular crosstalk and contributing to innovative therapeutic solutions for inflammatory and fibrotic heart disease.